The Neurological Rationale for a Comprehensive Clinical Protocol Using Applied Kinesiology Techniques

Walter Schmitt, D.C, D.I.B.A.K

ABSTRACT

A procedural protocol for the application of applied kinesiology techniques is presented. It is based on neurological and biochemical principles and thirty years of clinical observations of comparative applications of techniques. Short summaries of each section are included prior to the section to enable a brief review of the information.

SYTEMIC ENDOCRINE EFFECTS

14. Does TLR Strengthen as expected?

  1. No: Identify and Treat Appropriate Endocrine Chapman’s Reflex
  2. Yes: Check for Endocrine Related Muscle Weakness – Treat Appropriately

    15. Does Rubbing Adrenal Chapman’s Reflexes cause Pituitary Chapman’s Reflex to TL?

    1. Yes: IRT to Adrenal Chapman’s Reflexes with Offender

    16. Does Adrenal Challenge (Pinching) Induce Adrenal Muscle Weakness? If Yes:

    1. TL to Adrenal Chapman’s Reflexes – If Strengthens: Rub Reflexes
    2. TL to Pituitary Chapman’s Reflex – If Strengthens: Go to 15a

    17. Does Ligament Stretch Cause Muscle Weakness?

    1. Yes: Rub Adrenal Chapman’s Reflexes

    18. Test Endocrine Related Muscles – Identify and Treat Primary Chapman’s Reflex

    1. Test PMS (Liver) and TFL (Colon) – Treat Primary Chapman’s Reflex

    19. Test PMS – Rub and Pinch Liver VRP area – If Positive:

    1. Test Liver Detoxification Nutrients
    2. Challenge Liver Chapman’s Reflex with Offenders – If Positive:

            i. IRT Liver Chapman’s Reflex with Offenders c. Challenge PMS with Cholesterol – If Weakens: Go to 8c
            i. Rub Liver Chapman’s Reflex with Cholesterol in Mouth.

    20. Pinch Pancreas VRP & Test Biceps Brachii (or Other Upper Limb Flexor) – If Weakens:

    1. Test Chromium, Vanadium, Zinc, Pancreas Tissue, Sesame Seed Oil
    2. Challenge Pancreas Chapman’s Reflex with Offender – Offenders include:

            i. Milk, Cortisol, Bad Fats, NE, Other Allergens

    Summary: Identify the need for increasing or decreasing endocrine function. If indications are for both – correct toward increasing the sluggish organ function first. Any excessive hormone may be related to either over production or faulty liver detoxification. Liver assessment must include GI tract (especially large intestine) evaluation. Many endocrine problems include hyperinsulinism that must be addressed in conjunction with other endocrine dysfunction.

    Injuries (especially those causing need for IRT or NSB) are stressful to the body and many endocrine (especially adrenal) indicators will be present until the sources of nociception are corrected. Nociception drives the ascending spinothalamic tract (part of the anterolateral spinal tracts) that impacts the hypothalamus causing the HPT-pituitary-adrenal axis to be stimulated to produce increased cortisol levels. In a similar fashion, nociception that reaches the HPT will create a sympathetic fight or flee reaction. This is why we never assess endocrine (or autonomic) function until injury patterns are corrected. Since inflammatory mediators depolarize nociceptors and drive the nociceptive pathways, corrections to the basic chemistry of inflammation (EFA, allergies, antioxidants) must also be addressed. Histamine is a powerful adrenal stimulant that must be moderated prior to endocrine evaluation.

    As mentioned previously, Goodheart observed that muscles would respond differently to right brain activity (e.g., humming a tune) and to left brain activity (e.g., counting.) 12 He taught that right brain activity would often respond to therapies that increased steroid activity. Similarly, he noted that muscles that responded to left brain activity (e.g., counting) would often 22 respond the therapies that increased thyroid activity. Right-left brain imbalances of this nature can contribute to emotional stress (and cognitive) disturbances and the correction of endocrine imbalances at this point will set the stage for effective assessment of emotional recall issues later in the protocol.

    14. Does TLR Strengthen as expected?

    1. No: Identify and Treat Appropriate Endocrine Chapman’s Reflex
    2. Yes: Check for Endocrine Related Muscle Weakness – Treat Appropriately

    Correction of sluggish immune system activity is necessary prior to endocrine correction to prevent any further inhibition of already suppressed immune tissues by increased adrenal glucocorticoid activity resulting from endocrine balancing efforts. On a neurological level, correcting injuries, TMJ and cranial problems (whether primary mechanical problems or secondary to immune system imbalances) is necessary prior to addressing endocrine dysfunction. These corrections will restore cerebellar afferent and efferent pathways enough to allow a clear assessment of tonic labyrinthine reflex (TLR) activity.

      Hypofunction of an endocrine organ (adrenal, thyroid, reproductive) will be seen to cause an inappropriate muscular response to changes in head position relative to gravity. The first step in endocrine assessment is testing to see if the TLR are operating properly by testing inhibited muscles with the head in a position for TLR to facilitate those muscles. These reflexes operate primarily via the inferior vestibular nuclei, which receive direct input from the cerebellum. Failure of the TLR tells us that there is a under functioning endocrine organ, as long as other pathways to the vestibular nuclei are not interfering with its descending output.

      Although not often mentioned, when expected muscles do not strengthen with the left ear down TLR pattern, it is always indicative of a low steroid function, either adrenal or reproductive, although most often adrenal. When the right ear down TLR pattern does not cause the expected muscle response, it is indicative of a relative hypothyroid state. However, these right and left correlations can be relied upon only if the patient has first had patterns of switching corrected (which is another reason that we place switching prior to the assessment of endocrine function in our protocol.)

      15. Does Rubbing Adrenal Chapman’s Reflexes cause Pituitary Chapman’s Reflex to TL?

      1. Yes: IRT to Adrenal Chapman’s Reflexes with Offender.

      16. Does Adrenal Challenge (Pinching) Induce Adrenal Muscle Weakness? If Yes:

      1. TL to Adrenal Chapman’s Reflexes – If Strengthens: Rub Reflexes
      2. TL to Pituitary Chapman’s Reflex – If Strengthens: Go to 15a

      17. Does Ligament Stretch Cause Muscle Weakness?

      1. Yes: Rub Adrenal Chapman’s Reflexes

      In addition to hypoadrenia, the other adrenal patterns assessed (hyperadrenia and ligament stretch adrenal stress syndrome - LSASS) should be tested only after the immune system’s effects on the hypothalamus are resolved. The HPT drives the mesencephalon wherein the pattern generator cells of the parabrachial nucleus create “centering the spine” patterns including left convex lateral flexion (i.e., head and feet to right) associated with hyperadrenia (and increased reproductive steroids) and the right convex lateral flexion (i.e., head and feel to left) associated with increased thyroid activity. With the HPT-mesencephalon pathway free from immune interference (and other body chemistry problems previously addressed) we can accurately assess lateral flexion patterns.

      Correction of endocrine dysfunction will center the spine and make adjustments easier (on both the doctor and the patient) and longer lasting. LSASS must be ruled out prior to any spinal adjusting, especially in patients with a history of poor response to manipulation.

      18. Test Endocrine Related Muscles – Identify and Treat Primary Chapman’s Reflex

      1. Test PMS (Liver) and TFL (Colon) – Treat Primary Chapman’s Reflex

      Understanding endocrine function depends on grasping the concepts of endocrine interaction. At this point in our assessment, we must consider the following organs: adrenal, thyroid, reproductive, pituitary, and pineal. Immediately following we will address the pancreas and insulin/glucose metabolism that also plays an integral role. We know that a hormone abnormality is due to either too much or not enough of that hormone. If we have excessive hormone activity, we will be led to address the over production by treating the Chapman’s reflex of that organ using IRT with an offender. (Other endocrine assessment and treatment procedures that are not covered in this protocol may be used at this point as well as long as both hypo and hyper function of each organ are considered.)

      We may find one endocrine organ that is primary by cross-check TL. If found, this is the gland to which we address nutritional and manipulative efforts at this visit. We have already assessed the biochemical pathways (CAC, ETC) for energy production that are necessary for any organ’s optimal function, so we proceed with assessing hormones, glandulars, and herbals that will be most appropriate for support of the patient’s system in the now present state.

      Excess hormone can also be due to decreased breakdown / detoxification of the substance that primarily takes place in the liver. We recognize that, in the most general sense, the liver’s main job is to detoxify the bowel. So we must assess liver – large intestine function and interaction at this point. By this time we will have identified EFA and antioxidants required for proper liver function as well as B vitamins in the CAC and other substances needed for ATP synthesis that are also necessary for liver detoxification pathways. Therefore, we proceed by evaluating the interaction of the liver and gut, and their interaction with the endocrine system.

      Most often, cross check TL will identify a primary endocrine gland and a primary organ (liver and large intestine.) Sometimes, however, there will be one organ that, when its VRP is stimulated or its Chapman’s reflex is TLed, will resolve all of the other related muscle inhibitions. It is important to recognize that unfriendly flora in the colon can produce glucuronidase enzymes that break off (deconjugates) estrogen from its conjugation with glucuronic acid, allowing it to be reabsorbed into the portal circulation and return to the liver. When the liver or the large intestine are involved, it is sometimes necessary to jump ahead in the protocol (to the next step for liver detoxification or to the next section for the large intestine) to obtain optimal results.

      As previously mentioned, centering the spine patterns reflect endocrine and autonomic functions, presumably mediated via hypothalamic connections to the parabrachial nucleus pattern generator cells in the mesencephalon. Spinal flexion is, of course, associated with sympathetic “fight or flee” activity that is often described as the scared cat arching its back. Conversely, parasympathetic activity is seen in the person who is “laid back.” Lateral flexion patterns relate as mentioned above: Left convex lateral flexion represents increased steroid activity (adrenal and reproductive steroids) and the right convex lateral flexion represents increased thyroid activity. Right and left spinal torques are generated by gait patterns that are produced by increased pituitary / decreased pineal (right foot forward gait) or increased pineal / decreased pituitary (left foot forward gait) functions.

      Many systemic muscular patterns will resolve instantly following one specific endocrine correction related to centering the spine. Of course, adaptations to injuries will also cause 24 centering the spine problems, but these postural and visual (such as EID) responses to trauma will have long since been corrected by IRT, NSB, and/or SP techniques.

      Too often doctors work piece-meal at balancing structural function by addressing one local problem after another when there is a single underlying systemic centering the spine problem at the root of all of the individual muscle imbalances. One example of this is body into distortion (BID) patterns that are resolved upon making corrections related to centering the spine.

      Another example of this is the use of percussion techniques to many areas of the body to achieve normal spinal and extremity ranges of motion. Although the multiple application percussive treatment is effective, it is time consuming compared to the simple centering the spine corrections. It is also questionable whether or not the multiple percussion therapies achieve changes in the underlying systemic endocrine imbalances (or injuries) that have thrown the spine off-center in the first place. At this point, the use of percussion therapies to multiple areas is appropriate

      19. Test PMS – Rub and Pinch Liver VRP area – If Positive:

      1. Test Liver Detoxification Nutrients
      2. Challenge Liver Chapman’s Reflex with Offenders – If Positive:

              i. IRT Liver Chapman’s Reflex with Offenders c. Challenge PMS with Cholesterol – If Weakens: Go to 8c
              i. Rub Liver Chapman’s Reflex with Cholesterol in Mouth.

      In fact, a complete liver evaluation may or may not be necessary to correct endocrine imbalances. There may not even be any endocrine significance, but under any circumstances, if the liver VRP is active, now is the time to do a more in depth evaluation. Assessing liver detoxification may include screening with a number of related nutrients, most of which are needed for other important functions as well. Recurrence of liver VRP activity should include in-depth evaluation of the potential nutrients related to the faulty pathway, and this alone will cover most, if not all of the nutrient needs of your patient. The few exceptions will be picked up in the next step when looking at the pancreas or the next section when looking at the GI tract.

      Prior to checking the liver for hormone detoxification, we first want to improve any sluggish hormone production. Following treatment (rubbing Chapman’s reflexes) to correct an endocrine hypofunction, the levels of circulating hormone would be expected to rise. Therefore, the time to evaluate liver function for hormone detoxification is following endocrine stimulation. The liver might not have any difficulty keeping up with a lowered level of circulating hormones, but the normal circulating levels might challenge a low liver reserve. Therefore, we evaluate liver function after finding, and often treating, the primary endocrine gland.

      The same may be said for immune function. It is obvious that we would want to correct immune function prior to checking the liver for cytokine excess that is part of our liver evaluation. If the liver is incapable of keeping up with the demands of detoxifying normal cytokine production levels, and if the immune system is sluggish, the body may not show cytokine excess as a problem. The earlier correction of sluggish immune activity by stimulation of immune Chapman’s reflexes will subject the liver to increased cytokine levels thereby increasing the demand on the liver’s abilities to handle these substances. In the presence of increased immune function the liver may now be appropriately evaluated. Glycine, which is necessary for cytokine metabolism, is also one of the nutrients necessary for cholesterol metabolism and other liver detoxification. Glycine is also an important inhibitory NT. Be sure to check for glycine and its cofactors at this point if there is a cytokine problem.

      20. Pinch Pancreas VRP and Test Biceps Brachii (or Other Upper Limb Flexor)–If Weakens:

      1. Test Chromium, Vanadium, Zinc, Pancreas Tissue, Sesame Seed Oil
      2. Challenge Pancreas Chapman’s Reflex with Offender – Offenders include:

              i. Milk, Cortisol, Bad Fats, NE, Other Allergens

      The placement of pancreas endocrine assessment at this point is based as much on clinical observation as it is on biochemical sense. However, since insulin has enzyme blocking effects that impact steroid hormone synthesis, it must be included in the overall evaluation of the endocrine system.

      Major offenders to the pancreas are allergens and bad fats. In that light we may have already corrected some or all of the pancreas stress when we addressed allergy-related problems and EFA metabolism. Another common pancreas stressor (driving it to hyper function) is cortisol. So it is only appropriate that we reserve pancreas evaluation until after adrenal function is normalized to get a clearer picture of cortisol’s effects. Often, treating a cortisol problem will reveal a pancreas problem not apparent prior to adrenal correction.

      When dealing with a patient with joint problems, it may be wise to skip from number 8.d. in the protocol to this step and then return to Step 9. Glucosamine and glucuronic acid, necessary for the production of the mucopolysaccharides that make up connective tissue (hylauronic acid) and cartilage (chondroitin sulfate), require proper glucose metabolism. Glucose must enter the cell efficiently, which depends on insulin, and it must be metabolized properly via glycolysis (the Embden-Myerhof pathway) in order to be available for polymerization into the appropriate mucopolysaccarides.

      Insulin has a significant effect on the autonomic nervous system. Insulin increases sympathetic outflow (which is one of the reasons hyperinsulinism patients often present with hypertension, many times misdiagnosed as “idiopathic hypertension” when it is really secondary to the sympathomimetic effects of the excess insulin.) The neurological SYM response to increased insulin includes the spinal flexion pattern. Spinal flexion is usually accompanied by a spinal torque (gait) pattern as part of normal spinal coupled motion. Repetitive gait in the hyperinsulinism patient frequently leads to a spinal gait torque pattern that results in an iliolumbar ligament IRT pattern. Correcting the hyperinsulinism pattern normalizes the SYM spinal flexion and its concomitant spinal torque rotation. Certainly, if present, this should be addressed prior to checking the iliolumbar ligament as well as prior to administering any spinal and pelvic adjusting procedures. More will be said of this pattern in the discussion regarding gait.

      Bilateral upper extremity symptoms n any joint from the shoulders to the fingers are often secondary to hyperinsulinism. The excess response of pancreas insulin production is accompanied by a bilateral triceps over facilitation. Secondary inhibition of the biceps and/or other upper limb flexors results in bilateral upper limb symptoms which are often mistakenly treated as local problems.

      Hyperinsulinism patients will often need treatments to both decrease the pancreas response (IRT to the Chapman’s reflex with an offender) as well as to increase a tired pancreas by rubbing Chapman’s reflex. The latter will be identified when evaluating gait following a challenge of the pancreas VRP.

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