A procedural protocol for the application of applied kinesiology techniques is presented. It is based on neurological and biochemical principles and thirty years of clinical observations of comparative applications of techniques. Short summaries of each section are included prior to the section to enable a brief review of the information.

12. Does Rebreathing in a Paper Bag Strengthen?

1. If Yes: Check Citric Acid Cycle & Electron Transport Chain Nutritional Factors

Summary: CAC activity is necessary for carbon dioxide production for eventual synthesis of bicarbonate ion, which is necessary for CSF production and normal cranial bone function. CAC and ETC create ATP production for cellular function including maintenance of membrane polarization via membrane ion pumps. Several NTs are dependent on CAC function including glutamate, GABA, and aspartat

As previously mentioned, we check for CAC activity following other systemic nutritional and structural factors rather than before. Inflammatory activity of cytokines and the production of nitric oxide cause a truncation of the CAC, and any attempt to correct CAC function is like painting over rust until the underlying source of inflammation (from EFA, allergies and hypersensitivities, and/or antioxidant deficiency) is negated.

Goodheart wrote many years ago about using zinc for recurrent cranial faults. 18 Zinc catalyzes the carbonic anhydrase enzyme for the reaction between H2O and CO2 to produce carbonic acid (H2CO3) which then dissociates into hydrogen ions and bicarbonate ions. The bicarbonate ions are necessary for the production of cerebrospinal fluid (CSF) as well as many other functions in the body (e.g., kidney, lung.) Goodheart hypothesized that the lack of bicarbonate interfered with adequate CSF synthesis that then led to cranial faults as adaptations to the change in CSF pressure and flow.

To continue this train of thought, even more fundamental than zinc in this process is the availability of CO2. CO2 is a major byproduct of CAC activity. Inadequate CAC function will have an even more fundamental effect on CSF production than a need for zinc. Of course, there could be a need for both improved CAC activity and zinc, but zinc without the source of CO2 will be ineffective. The other source of CO2 is from vitamin B-6 dependent decarboxylation reactions throughout the body. B-6 is essential for the production of most NTs and its need will be found when CO2 strengthens and the CAC assessment finds no other CAC need.

So checking for CAC function at this juncture is also related to the possible recurrence of cranial faults that could indicate a problem with CAC activity (and/or zinc.) It is important to note that the excitatory NT glutamate and the inhibitory NT GABA are synthesized from alphaketoglutaric acid which is generated by the CAC. Sluggish CAC activity will interfere with the availability of these two important NTs and alter both sensory and motor responses to our other treatment procedures. Hence, it is a good time to make sure that these pathways are functioning adequately.

It is important to note that CO2 combines with ammonium ion to generate the urea cycle, the major source of ammonia waste for the body. Inability to rid the body of ammonia results in 19 hyperammonemia, resulting in changes in NT synthesis as ammonia groups are added to certain amino acid based NTs converting them into different substances altogether. For example, glutamate will become GABA, but in the presence of excess ammonia, it will be converted to glutamine and GABA will become deficient. Similarly, aspartate can be converted to asparagines and its NT function negated.

Speaking strictly about energy production rather than the other ramifications of CAC problems, we see that this is also the proper placement of CAC and ETC assessment. As stated previously, the need for CoQ10 could be performed earlier. But the CAC and the ETC are a continuum in the oxidative phosphorylation process for the production of ATP. ATP provides energy for every cellular energy-using function: muscles, organs, and neurons. Once we have eliminated sources of CAC blockade (inflammation and/or allergy causing cytokines and nitric oxide) we can look to the possible need for B vitamins and manganese (and sometimes iron) necessary for efficient CAC activity.

It is important to recognize, from a neuron metabolism point of view, that all membrane receptor pumps (e.g., sodium-potassium pumps) that maintain neuron membrane polarization at a negative resting potential are energy (ATP) using mechanisms. If we want to have optimal depolarization (neuron firing) and repolarization, we must have adequate ATP formation, and this depends on CAC and ETC efficiency.

As mentioned previously, several neurotransmitters are dependent on the CAC including glutamate, aspartate, and GABA. Glutamate and Aspartate are excitatory NTs and GABA is the most important inhibitory NT in the CNS. Drugs that function as GABAergic agents include anti-anxiety drugs such as the benzodiazepines. In patients with low GABA, emotional treatments aimed at reducing anxiety will be short-lived, if successful at all. So, we address the production of GABA prior to treating emotional recall related disorders. In similar fashion, we have already dealt with the histamine produced from allergic responses above. Excess histamine, related to depression and other emotional responses, should be addressed prior to focusing on emotional recall techniques.

Similarly, in addition to the CAC nutrients, other nutrients necessary for NT formation (e.g., folic acid, B-6, iron) have also been covered to this point so that the patient will be able to maintain emotional as well as other NT based corrections. If the patient is treated for an emotional stress recall problem (or other NT dependent problem) in the same treatment session that a NT related nutrient need was identified, it is important to have patient insalivate and ingest the nutrient during the treatment session to reinforce the effects of the NT dependent treatment.

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