A procedural protocol for the application of applied kinesiology techniques is presented. It is based on neurological and biochemical principles and thirty years of clinical observations of comparative applications of techniques. Short summaries of each section are included prior to the section to enable a brief review of the information.

31. Check Gait (backward step first)

    1. If Gait Testing Facilitation/Inhibition ABNORMAL

        i. Check Iliolumbar Ligament or Pelvic Category or Foot/Ankle Subluxation

    2. If Gait Testing Facilitation/Inhibition NORMAL

        i. Pinch Pancreas VRP – If Pinching VRP Disrupts Gait: Test Nutrients
        ii. Rub Pancreas Chapman’s Reflex
        iii. Pinch Other VRP’s – If Pinching Disrupts Gait: Rub Chapman’s Reflex

Summary: After all other corrections have been made, assessing the patient in the right and left gait patterns will assure that normal gait will not wind down the effects of the treatment. Gait patterns might remain in the presence of uncorrected structure, uncorrected endocrine hypo function, and/or residual visceral effects, most notably from hyperinsulinism.

There may still be a disturbance in gait, even after addressing all of the previous protocol steps. If there is a major structural fault (pelvis, spine, foot/ankle) or IL ligament that has been missed, it will cause a disturbance in gait that must now be corrected. However, residual gait disturbance is most commonly due to the need for performing the second step in the correction of hyperinsulinism problems – the need for rubbing Chapman’s reflex for the pancreas. It may also be due to residual dysfunction in any other viscera including the endocrine system.

The presence of endocrine hypo function can be seen at this point by assessing TLR patterns during gait. During gait testing, tilting the head so that the ear is pointed toward the ground on the side of extensor (e.g., latissimus dorsi) inhibition should override the gait-induced weakness. If this does not occur, TL to one of the endocrine Chapman’s reflexes will temporarily restore this TLR function. Rubbing the Chapman’s reflex identified will correct the disturbance noted on challenging gait with TLR activity.

Hyperinsulinism is extremely common in the patients chiropractors see, probably due, in part, to the spinal flexion effects of insulin and the adaptive spinal torque (gait) stresses. The spinal torque will create residual muscle tightness (and often pain) even after all else is corrected. Long term asymmetrical sensory activity from the spine and extremities can create a cerebral cortical hemisphericity that may require neurological assessment and treatment. However, more often, hyperinsulinism will mimic a hemisphericity pattern, and treating Chapman’s pancreas reflex at this point can resolve the imbalance by correcting the source.

Many residual muscular symptoms, anywhere in the body, will resolve upon rubbing the pancreas Chapman’s reflex in hyperinsulinism patients. This is due to relieving the remaining insulin induced SYM spinal stress and consequent spinal adaptations.

For these reasons it is recommended that gait be checked on any patient prior to the end of the treatment session, regardless of which procedures were performed previously during that session. This will prevent the patient from walking out of the office with a gait asymmetry that will wind down all of the previous mechanical and neurological corrections, including recurrence of imbalances associated with cortical hemisphericity.

Copyright 2019- QA Home Study- All rights Reserved